Figure 1a: Effects of DG on cerebral blood flow and systemic blood pressure and in rats subjected to global brain ischaemia-reperfusion injury. DG had no significant effect on cerebral blood flow and systemic blood pressure of rats subjected to global brain ischaemia-reperfusion. One-way ANOVA, ***P<0.001. The data are expressed as the mean±SD, n=6-7

Figure 1b: Effects of DG on cerebral blood flow and systemic blood pressure and in rats subjected to global brain ischaemia-reperfusion injury. DG had no significant effect on cerebral blood flow and systemic blood pressure of rats subjected to global brain ischaemia-reperfusion. One-way ANOVA, ***P<0.001. The data are expressed as the mean±SD, n=6-7

Figure 2a: Effects of DG on malondialdehyde (MDA) and nitrite levels in rat brains subjected to global ischaemia-reperfusion injury. DG inhibited the increase in MDA (A) and nitrite levels (B) in the ischaemic brains. One-way ANOVA, *P<0.05, **P<0.01, ***P<0.001. The data are expressed as the mean±SD, n=5-7

Figure 2b: Effects of DG on malondialdehyde (MDA) and nitrite levels in rat brains subjected to global ischaemia-reperfusion injury. DG inhibited the increase in MDA (A) and nitrite levels (B) in the ischaemic brains. One-way ANOVA, *P<0.05, **P<0.01, ***P<0.001. The data are expressed as the mean±SD, n=5-7

Figure 3a: Effects of DG on anti-oxidant enzymes in rat brains subjected to global ischaemia-reperfusion injury. DG restored superoxide dismutase (SOD) (A), catalase (CAT) (B), and reduced glutathione (GSH) (C) levels in the ischaemic brains. One-way ANOVA, *P<0.05, **P<0.01, ***P<0.001. The data are expressed as the mean±SD, n=6-7

Figure 3b:Effects of DG on anti-oxidant enzymes in rat brains subjected to global ischaemia-reperfusion injury. DG restored superoxide dismutase (SOD) (A), catalase (CAT) (B), and reduced glutathione (GSH) (C) levels in the ischaemic brains. One-way ANOVA, *P<0.05, **P<0.01, ***P<0.001. The data are expressed as the mean±SD, n=6-7

Figure 3c: Effects of DG on anti-oxidant enzymes in rat brains subjected to global ischaemia-reperfusion injury. DG restored superoxide dismutase (SOD) (A), catalase (CAT) (B), and reduced glutathione (GSH) (C) levels in the ischaemic brains. One-way ANOVA, *P<0.05, **P<0.01, ***P<0.001. The data are expressed as the mean±SD, n=6-7

Figure 4a: Effects of DG on indexes of brain injuries induced by focal (MCAO)-reperfusion injury. Representative photos of TTC staining of brain slices show that DG reduced the cerebral infarct size (A). DG also significantly reduced infarct weight (B) and neurological deficit (C) in the ischaemic brains. One-way ANOVA, *P<0.05, **P<0.01, ***P<0.001. The data are expressed as the mean±SD, n=5-6

Figure 4b:Effects of DG on indexes of brain injuries induced by focal (MCAO)-reperfusion injury. Representative photos of TTC staining of brain slices show that DG reduced the cerebral infarct size (A). DG also significantly reduced infarct weight (B) and neurological deficit (C) in the ischaemic brains. One-way ANOVA, *P<0.05, **P<0.01, ***P<0.001. The data are expressed as the mean±SD, n=5-6

Figure 4c: Effects of DG on indexes of brain injuries induced by focal (MCAO)-reperfusion injury. Representative photos of TTC staining of brain slices show that DG reduced the cerebral infarct size (A). DG also significantly reduced infarct weight (B) and neurological deficit (C) in the ischaemic brains. One-way ANOVA, *P<0.05, **P<0.01, ***P<0.001. The data are expressed as the mean±SD, n=5-6